What is Clomifene Citrate ?
Since clomid has ability to bind to the estrogen receptors, much like nolvadex, in male users who run estrogenic compounds such as testosterone, dianabol, and deca durabolin, it can be used as gynecomastia treatment (anti-gyno).
Clomid (Clomifene Citrate) is a powerfully effective anti-estrogen officially classified as a Selective Estrogen Receptor Modulator (SERM). In many ways, it is very similar to another popular SERM in Nolvadex (Tamoxifen Citrate). It is also one of the most commonly used SERM’s by anabolic steroid users. But it is not an anabolic steroid but can be used to combat estrogenic side effects sometimes caused by anabolic steroids. It can also be used as a Post Cycle Therapy (PCT) medication in order to stimulate suppressed testosterone production due to anabolic steroid use. PCT use of Clomid is the most common purpose and most beneficial point of use for the anabolic steroid user.
Clomiphene Citrate half-life is around 5 to 7 days, so you can take an average 6 day half-life as a reference point.
As an anti-estrogen for on cycle steroid use, Clomid is fairly effective at staving off gynecomastia. It will not reduce estrogen levels or inhibit the aromatization process, but in many cases, binding to the receptors is enough protection for many men. More importantly, when using Clomid for this purpose it can also have a positive impact on cholesterol levels. While an anti-estrogen, Clomid actually acts as estrogen in the liver, which in turn will promote healthier cholesterol levels.
For many men, especially hardcore anabolic steroid users, Clomid is not enough for estrogenic protection. In this case, an Aromatase Inhibitor (AI) like Arimidex (Anastrozole) or Femara (Letrozole) will be needed. AI’s actively inhibit the aromatase process and will see serum estrogen levels reduced. By far they are the most effective at combating gynecomastia and will have more success in combating water retention.
Then we’re left with the effects of Clomid as they pertain to PCT. When we supplement with anabolic steroids, this will suppress natural testosterone production. The rate of suppression will be dependent on the steroids being used and to a degree the total dosing, but it will generally be substantial. Further, natural testosterone recovery is dependent on no prior low testosterone condition existing and assumes that no severe damage was done to the Hypothalamic-Pituitary-Testicular-Axis (HPTA) through improper anabolic steroid use.
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