What is Clomifene Citrate ?
Male bodybuilders who cycle anabolic androgenic steroids (AAS) have found that the same mechanisms of increased LH (luteinizing hormone) can be taken advantage of to increase total testosterone levels. This is why clomid is popular in post cycle therapy (PCT) among those who cycle anabolic steroids. Since clomid has ability to bind to the estrogen receptors, much like nolvadex, in male users who run estrogenic compounds such as testosterone, dianabol, and deca durabolin, it can be used as gynecomastia treatment (anti-gyno). However, clomid is a fairly weak anti-gyno aid, and is rarely used for that purpose. Due to its ability to boost testosterone levels, it has been put on the WADA (world anti-doping agency) list of illegal doping agents in sport.
Clomid (Clomifene Citrate) is a powerfully effective anti-estrogen officially classified as a Selective Estrogen Receptor Modulator (SERM). In many ways, it is very similar to another popular SERM in Nolvadex (Tamoxifen Citrate). Clomid first gained worldwide attention in the early 1970’s as a strong fertility aid and is still used for that purpose today. It is also one of the most commonly used SERM’s by anabolic steroid users. No, it is not an anabolic steroid but can be used to combat estrogenic side effects sometimes caused by anabolic steroids. It can also be used as a Post Cycle Therapy (PCT) medication in order to stimulate suppressed testosterone production due to anabolic steroid use. PCT use of Clomid is the most common purpose and most beneficial point of use for the anabolic steroid user.
Clomiphene Citrate half-life is around 5 to 7 days, so you can take an average 6 day half-life as a reference point.
In a therapeutic setting as a fertility aid, the effects of Clomid are simple. The woman is having difficulty becoming pregnant, Clomid enhances the chance of conception and pregnancy occurs. It doesn’t always work and we’ll look at the process in the administration section, but it will greatly increase the odds of conception and is fairly successful.
As an anti-estrogen for on cycle steroid use, Clomid is fairly effective at staving off gynecomastia. It will not reduce estrogen levels or inhibit the aromatization process, but in many cases, binding to the receptors is enough protection for many men. In fact, while there are stronger protective agents, many men would be surprised as to how well a SERM like Clomid can work if they would give it a chance. More importantly, when using Clomid for this purpose it can also have a positive impact on cholesterol levels. While an anti-estrogen, Clomid actually acts as estrogen in the liver, which in turn will promote healthier cholesterol levels. This can be very beneficial to the anabolic steroid user as anabolic steroid use is notorious for promoting unhealthy cholesterol levels.
For many men, especially hardcore anabolic steroid users, Clomid is not enough for estrogenic protection. In this case, an Aromatase Inhibitor (AI) like Arimidex (Anastrozole) or Femara (Letrozole) will be needed. AI’s actively inhibit the aromatase process and will see serum estrogen levels reduced. By far they are the most effective at combating gynecomastia and will have more success in combating water retention. In fact, SERM’s like Clomid often do very little to combat water retention. However, in many cases, many performance athletes would do a better job controlling water retention if they did a better job controlling their diet. In off-season bulking plans this requires excess calories to grow. However, many take it too far, especially carbohydrate consumptions, and this will cause you to hold water with or without anabolic steroid use. Add in aromatizing steroids to the equation and water retention will be even greater. Further, AI’s while effective will have a negative impact on cholesterol, which leads us to only one sane conclusion. Control your diet and if you can control gynecomastia with a SERM like Clomid it should be your first choice.
Then we’re left with the effects of Clomid as they pertain to PCT. When we supplement with anabolic steroids, this will suppress natural testosterone production. The rate of suppression will be dependent on the steroids being used and to a degree the total dosing, but it will generally be substantial. For this reason, most men will always include exogenous testosterone in their steroid cycles. In fact, it’s not uncommon for it to be the only steroid used. This will protect the individual from a low testosterone condition while on cycle. Unfortunately, once steroid use comes to an end natural testosterone levels will be very low. Natural testosterone production will begin again on its own, but it will take a significant amount of time for levels to reach their previous high state. In fact, total recovery can take months to even a year. Further, natural testosterone recovery is dependent on no prior low testosterone condition existing and assumes that no severe damage was done to the Hypothalamic-Pituitary-Testicular-Axis (HPTA) through improper anabolic steroid use.
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